Angioedema is defined as ” subcutaneous tissues and/or submucosal tissues circumscribed non-pitting edema affecting lips, face, neck, and extremities oral cavity, larynx, and gut.”
It becomes life-threatening when it involves the larynx, while intestinal angioedema is painful and mimics acute abdomen.
Classification of angioedema
Acquired
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Allergic (histaminergic angioedema) associated with anaphylaxis,
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Non-allergic (non-histaminergic angioedema), presenting isolated or in combination with urticaria,
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Drug-induced (e.g. allopathic angiotensin-converting enzyme inhibitors and non-steroidal anti-inflammatory drugs),
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Complement-mediated secondary to acquired deficiency of C1-inhibitor (allopathic),
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Idiopathic which is subdivided into histaminergic and non-histaminergic (allopathic).
Any known Homeopathic medicine has no reaction(s) or side effects like angioedema etc.
Hereditary forms
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C1-Inhibitor deficiency divided into type 1 (lack of C1-inhibitor molecule) and typed 2 (dysfunctional C1-inhibitor molecule) with normal C1 inhibitor.
Etiology
Inherited (hereditary angioedema): Mutations in the gene encoding for C1-inhibitor cause hereditary angioedema and it is an autosomal dominant condition.
Acquired: lymphoproliferative disorders, autoimmune, neoplastic, infection and allopathic drug induced.
Epidemiology
A retrospective study showed angioedema was the second most common disorder after asthma for hospitalization in many countries.  Hereditary angioedema is a rare autosomal dominant condition. Hereditary angioedema affected females more severely compared to males.
Pathophysiology
In describing pathophysiology, angioedema is classified as histamine-mediated angioedema and bradykinin-mediated angioedema.
Histamine-mediated angioedema
Histamine-mediated angioedema is the most common and is secondary to mast-cells and basophil activation.
Bradykinin-mediated angioedema
Bradykinin-mediated angioedema (hereditary angioedema, acquired C1-inhibitor deficiency and angiotensin-converting enzyme inhibitor-associated angioedema). Allergic reactions and hives do not trigger this condition. C1-inhibitor is a regulator of complement and the contact system; if deficient or dysfunctional it causes activation of the contact system resulting in uncontrolled production of kallikrein leading to proteolysis of high-molecular-weight kininogen and bradykinin, leading to edema by increasing in vascular permeability. Bradykinin-mediated angioedema is seen in:
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C1 inhibitor deficiency: Excessive production of bradykinin,
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Angiotensin-converting enzyme inhibitor-associated angioedema: Decreased degradation of bradykinin,
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Histamine and bradykinin increase localized microvascular permeability,
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NSAID induced: cyclooxygenase 1 inhibitor affects arachnoid acid metabolism, leukotriene/prostaglandin binding to the receptor or may be IgE mediated.
Genetics:
Hereditary angioedema can be secondary to F12 gene, angiopoietin-1, and plasminogen or some unknown gene mutation.
C1 inhibitor is a serine protease inhibitor (SERPIN) C1 inhibitor deficiency is associated with SERPING 1 mutation or mutated genes which encode for metabolizing and functioning enzymes of bradykinin. Abnormal accumulation of C1 inhibitor in dominant harmful disease affects plasma levels of hereditary angioedema type 1.
History and Physical/Symptoms
The presentation can be acute or chronic.
Histaminergic angioedema
Symptoms can involve different systems including skin (Urticaria, flushing, pruritus), Respiratory (bronchospasm), GI symptoms (abdominal pain and vomiting). Onset is within 60 minutes of allergen exposure and may last for one to two days.
Etiologies: Drugs, foods, latex, and insect stings.
While comparing the bradykinin-mediated angioedema with histaminergic angioedema, the former has the following characteristics:
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It is not associated with urticaria,
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More severe and longer duration,
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Has associated abdominal symptoms.
Acquired angioedema related to angiotensin-converting enzyme (ACE) inhibitor
It clinically presents as angioedema without urticaria or itching. It can develop anytime but common in the first week of exposure. 
Nonsteroidal anti-inflammatory disease-induced angioedema
NSAID induced drug reactions include angioedema presenting with urticaria and facial swelling.
Hereditary angioedema
Hereditary angioedema begins in childhood or young adulthood, gets worse at puberty and presents as recurrent episodes of swelling or abdominal pain. Patients can develop prominent prodromal symptoms like erythema marginatum (erythematous, serpentine, non-pruritic rash). An acute attack takes one day to peak and resolves in two to three days.
Acquired angioedema with C1 inhibitor deficiency
Acquired C1 inhibitor deficiency presents similarly to hereditary angioedema. However, the low C1 inhibitor in many cases is from an underlying lymphoproliferative disorder which increases protein consumption and an antibody against C1-INH causing overproduction of bradykinin.
Physical examination
Vital signs, level of consciousness, as well as a thorough skin, head, neck, respiratory and abdomen evaluation should be completed.
General monitoring of angioedema in the Emergency room include:
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Oxygen saturation,
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Cardiac status.
Evaluation
The initial evaluation in the Emergency room
A specific drug and family history is needed along with screening blood work for C4 for hereditary angioedema and tryptase for angioedema with anaphylaxis. These labs when drawn during acute attacks are useful during follow-ups. In the case of anaphylaxis, (tryptase is normal in hereditary angioedema one and two but will be elevated in cases of anaphylaxis and other mast cell disorders associated with angioedema). Flexible fiberoptic laryngoscopy may be done to evaluate the involvement of tongue and larynx in patients with head, neck and upper airway symptoms.
Clear differentiation between histamine-induced vs. bradykinin-induced angioedema can be lifesaving.
Hereditary angioedema type 1: C1 inhibitor function low, C1-Inhibitor level low, and C4 level low
Hereditary angioedema type 2: C1 inhibitor function and C4 level will be low, but the C1 inhibitor level will be normal or high.
🩺 Confirm them by repeating the blood test.
Acquired C1-inhibitor deficiency: Low C1-inhibitor antigen and function and low C1q.
Differential Diagnosis
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Acute contact dermatitis,
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Drug rash with eosinophilia and systemic symptoms,
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Dermatomyositis,
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Morbus Morbihan,
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Superior vena cava syndrome,
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Subcutaneous emphysema,
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Orofacial granulomatosis,
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Hypocomplementemic urticarial vasculitis syndrome,
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Clarkson’s disease,
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Gleich’s syndrome,
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A cluster headache,
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Idiopathic edema.
Angioedema is one of the differential diagnoses in sudden onset of diffuse isolated edema.
C1 inhibitor hereditary angioedema can be misdiagnosed as familial Mediterranean fever.
Allopathic treatment / Management
Antihistamine, corticosteroids, and epinephrine are treatments of histaminergic angioedema. 
Treatment of Bradykinin-mediated angioedema is often resistant to standard therapies such as epinephrine, glucocorticoids or antihistamines.
Hereditary angioedema (On-demand treatment)
Pre-procedural (short-term) prophylaxis
Short-term prophylaxis before high-risk procedures in high-risk individuals. Anesthesiologists must be aware of guideline-based treatment for hereditary and acquired angioedema.
Long-term prophylaxis
C1 Inhibitor is the first-line long-term prophylaxis, while the androgens are used as second-line agents.
Hereditary angioedema with mutations in the F12 gene is treated as follows:
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Discontinuing trigger factors (estrogen-containing oral contraceptives, hormonal replacement therapy, angiotensin-converting enzyme inhibitors – all these only allopathic; Homeopathic has no side effects),
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Treating with plasma-derived C1 inhibitor for acute attacks. Preventing attacks with progestins, tranexamic acid, and danazol.
Acquired angioedema related to angiotensin-converting enzyme (ACE) inhibitor
Treatment is with antihistamines, epinephrine, and glucocorticoids. Care should be taken to stop the offending ACE inhibitor, and the patient should be not rechallenged with any of the ACE inhibitors in the future.
Acquired C1 inhibitor deficiency angioedema
Majority cases are asymptomatic and respond to immunochemotherapy Treatment of acute attacks with icatibant and plasma-derived C1 inhibitor concentrate, and prophylaxis is with rituximab with or without chemotherapy and splenectomy.
Special considerations:
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In pregnancy, the recommended therapy is plasma-derived nano-filtered C1-inhibitor, however, in acute episodes, bradykinin receptor antagonist Icatibant can be used as it has lower with no maternal and fetal adverse effects.
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In the pediatric population, the doses include 500 units (10-25 kg weighed patients), 1000 units and 1500 Units in patients weighing more than 25kg.
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Plasma-derived C1 esterase inhibitor is efficacious in pediatric patients below 12 years.
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Icatibant is a well-tolerated medication in the pediatric group and might have a role in treating angiotensin II receptor blocker induced angioedema.
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Recombinant human C1 esterase inhibitor for acute hereditary angioedema treatment has a persistent response for three days.
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C1 inhibitor is used in the acute management of hereditary angioedema associated pancreatitis.
Orolingual angioedema
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Patients with life-threatening orolingual angioedema who are treated with recombinant tissue plasminogen activator infusion, have a rapid response after using icatibant treatment.
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Concurrent use of angiotensin-converting enzyme inhibitors with dipeptidyl peptidase-4 inhibitors should be monitored closely as dipeptidyl peptidase-4 is also a major enzyme in the degradation pathway of bradykinin like an angiotensin-converting enzyme.
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Use of liquid steroids in patients with severe urticaria associated angioedema in a setting of severe dysphagia secondary to anaphylaxis.
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Use of omalizumab (Anti-immunoglobulin-E antibody) in Idiopathic non-histaminergic acquired angioedema, which is a rare disease resistant to antihistamines. 
Homeopathic treatment for Angioedema
Chemomilla
Throbbing headache in one-half of the brain. Inclined to bend head backward. Hot, clammy sweat on forehead and scalp. Ringing in ears. Earache, with soreness; swelling and heat driving patient frantic. Stitching pain. Ears feel stopped. Lids smart. Yellow sclerotic. Spasmodic closing of lids.
Cina Maritima
Intestinal irritation, worms and accompanying complaints. An irritability of temper, variable appetite, grinding of teeth, convulsions, with screams and violent jerking of the hands and feet. The Cina patient is hungry, cross, ugly, and wants to be rocked. Pain in shocks. Skin sensitive to touch. Pale, hot, with dark rings around eyes. Cold perspiration. White and bluish about the mouth. Grits teeth during sleep. Chorea like movements of face and hands.
Epigastric pain; worse, first waking in morning and before meals. Vomiting and diarrhea immediately after eating or drinking. Gagging cough in the morning. Violent recurring paroxysms, cough ends in a spasm. Periodic; returning spring and fall. Swallows after coughing. Gurgling from throat to stomach after coughing. Patient is afraid to speak or move for fear of bringing on paroxysm of coughing. After coughing, moaning, anxious, gasps for air and turns pale.
Senna (Cassia Angustifolia Vahl)
Universal swelling. Exhaustion. Unclean body swell. Sleep. Ebullition of blood disturbing the sleep. Sleeplessness, with cries and tossing. Colic. Flatulence. Anorexia Nervosa. Livid lips. Commissurae of lips covered with small burning vesicles. Repeated sneezing with temperature, exhaustion, and panting breathing. Hepatomegaly.
Sanguinaria Canadensis
Vaso-motor disturbances. Circumscribed redness of the cheeks, flashes of heat, determination of blood to head and chest, distention of temporal veins, burning in palms and soles. Burning sensations. Influenza. Phthisis. Sudden stopping of catarrh of respiratory tract followed by diarrhea. Burning in various parts. Flushed. Neuralgia: pain extends in all directions from upper jaw. Redness and burning of cheeks. Hectic flush. Fullness and tenderness behind angle of jaws.
Hay-fever. Ozaena, with profuse, offensive yellowish discharges. Nasal polyps. Coryza, followed by diarrhea. Chronic rhinitis; membrane dry and congested. Humming and roaring. Aural polypus. Throat swollen, dry and constricted. Unquenchable thirst. Burning, vomiting.
Filix Mas
Great feeling of weakness, trembling; cramp-like feelings in hands and feet. Somnolent, with spasmodic symptoms, ending in death. Blindness; first mydriasis and amaurosis with normal fundus; after few days, atrophy of optic nerve. Immobility of pupil. Itching of nose. violent dyspnea without cough, with stitches in region of heart and obscure symptoms of pericarditis. Pale face; blue rings round eyes. Nausea. Sick after taking food. Bloating of abdomen. Gnawing and boring in bowel.
Iodim
Sever fatigue, slightest effort induces perspiration. Enlarged lymphatic glands. Atrophy of all glandular structures, respiratory organs, circulatory system are especially affected. Acute exacerbation of chronic inflammation. Arthritis deformans. Acts prominently on connective tissue. Goiter. Abnormal vaso-constriction, capillary congestion followed by edema, ecchymosis, hemorrhages, and nutritive disturbances.
Acute catarrh of all mucous membranes, rapid emaciation, notwithstanding good appetite, and glandular atrophy. Acute affections of the respiratory organs. Pneumonia, rapid extension. Iodine is warm and wants cool surroundings.
Dulcamara
Hot days and cold nights towards the close of summer are especially favorable to the action of Dulcamara. It has a specific relation also to the skin, glands, and digestive organs, mucous membranes secreting more profusely while the skin is inactive. One-sided spasms with speechlessness. Paralysis of single parts. Congestive headache, with neuralgia and dry nose. Complete stoppage of nose. Stuffs up when there is a cold rain.
Thick, yellow mucus, bloody crusts. Earache, buzzing, stitches, and swelling of parotids. Swelling and indurated glands. Vesicular eruptions. Sensitive bleeding ulcers. Little boils. Red spots, urticaria, brought on by exposure, or sour stomach. Humid eruptions on face, genitals, hands, etc. Warts, large, smooth and palms. Anasarca. Thick, brown-yellow crusts.
Glycyrrhiza glabra
Neural disorders mainly ischemic stroke as well as the neurodegenerative diseases such as dementia and Alzheimer’s disease, and Parkinson’s disease. It has good anti diabetic activity. It has HMGB inhibitory function, reactive oxygen scavenging and anti-inflammatory activity, reducing chronic inflammatory conditions of air passage. Hyperlipidemia (an increase in one or more of the plasma lipids, including triglycerides, cholesterol, cholesterol esters, phospholipids and or plasma lipoproteins including very low-density lipoprotein and low-density lipoprotein along with reduced high density lipoprotein levels).
Glycyrrhiza glabra is a good supplement for treatment of dyslipidemia. Improves LDL, HDL, cholesterol. It has an inhibitory effect on of hepatic enzymes and possible liver toxicity.
Urtica Urens
Itching blotches. Urticaria, burning heat, with formication; violent itching. Consequences of suppressed nettle rash. Rheumatism alternates with nettle-rash. Burn confined to skin. Urticaria nodosa. Erythema, with burning and stinging. Burns and scalds. Chickenpox. Angioneurotic edema. Herpes labialis with sensation of heat and itching.
Bovista Lycoperdon
Scurf and crusts about nostrils and corners of mouth. Lips chapped. Bleeding of nose and gums. Cheeks and lips swollen. Acne worse (due to cosmetics). Weakness of all joints; clumsiness with hands, drops things. Weariness of hands and feet. Sweat in axillae, onion smell. Tip of coccyx itches intolerably. Moist eczema, formation of thick crusts. Itching of feet and legs. Edema in joints.
Blunt instruments leave deep impression on the skin. Urticaria on excitement, after morning walk, with rheumatic lameness, after bath, palpitation and diarrhea. Itching on getting warm. Pimples cover entire body; scurvy; herpetic eruptions. Pruritus ani.
Mercsolubilis
Tingling, burning, fetid eruptions on scalp. Loss of hair. Exostosis, with feeling of soreness. Scalp tense; oily sweat on head. Lids red, thick, swollen. Profuse, burning, acrid discharge. Floating black spots. Iritis, with hypopyon. Thick, yellow otorrhea; fetid and bloody. Otalgia, worse warmth of bed; at night sticking pains. Boils in external canal. Buboes. Orchitis. Nostrils raw, ulcerated; nasal bones swollen. Yellow-green, fetid, pus-like discharge. Coryza. Pain and swelling of nasal bones, and caries, with greenish fetid ulceration. Face pale, earthy, dirty-looking, puffy. Aching in facial bones, Syphilitic pustules on face.
Sweetish metallic taste. Bloody, viscid, fetid and coppery saliva. Trembling tongue. Gums spongy, recede, bleed easily. Tongue heavy, thick; moist coating; yellow, flabby, with ulcers. Glands swell every time patient takes cold. Throat bluish-red swelling. Constant desire to swallow. Ulcers and inflammation appearing at every change in weather. Stitches into ear on swallowing; fluids return through nose. Quinsy, with difficult swallowing. Sore, raw, smarting, burning throat. Complete loss of voice.
Agaricus Muscarious
Various forms of neuralgia and spasmodic affections, and neurotic skin troubles. Skin burning, itching, redness, and swelling. Pimples, hard, like fleabites. Miliary eruption, with intolerable itching and burning. Chilblains. Angioneurotic edema; rosacea. Swollen veins with cold skin. Circumscribed erythematous, papular/papules and pustular and edematous lesions. Facial muscles stiff; twitch; face itches and burns. Lancinating, tearing pain in cheeks. Neuralgia, as if cold needles ran through nerves or sharp ice touched them.
Double vision, dim and flickering. Asthenopia from prolonged strain, spasm of accommodation. Twitching of lids and eyeballs. Margins of lids red; itch and burn and agglutinate. Inner angles very red. Nose itching internally and externally. Spasmodic sneezing after coughing; sensitiveness. Fetid, dark, bloody discharge. Burning and smarting on lips. Herpes on lips. Twitching. Taste sweet. Aphthae on roof of mouth. Splinter like pains in tongue. Stitches along eustachian tube to ear. Feels contracted. Dryness of pharynx, swallowing difficult. Scratching in throat.
Arsenicum Album
Burning in eyes, with acrid lachrymation. Lids red, ulcerated, scabby, scaly, granulated. Edema around eyes. External inflammation, with extreme painfulness; burning, hot, and excoriating lachrymation. Corneal ulceration. Intense photophobia; better external warmth. Ciliary neuralgia, with fine burning pain. Skin within, raw and burning, itching, swellings; edema, eruption, papular, dry, rough, scaly; worse cold and scratching. Malignant pustules. Ulcers with offensive discharge. Anthrax. Poisoned wounds. Urticaria, with burning and restlessness. Psoriasis. Scirrhous. Icy coldness of body. Epithelioma of the skin. Gangrenous inflammations.
Offensive otorrhea. Roaring in ears, during a paroxysm of pain. Nose stopped up, sneezing without relief. Face swollen, pale, yellow, cachectic, sunken, cold, and covered with sweat. Expression of agony. Tearing needle-like pains; burning. Lips black, livid. Angry, circumscribed flush of cheeks. Throat swollen, edematous, constricted, burning, unable to swallow. Diphtheritic membrane looks dry and wrinkled.
Antracicum
A best medicine in epidemic spleen diseases and in septic inflammation, carbuncles and malignant ulcers, boils and boil-like eruptions, acne etc. Terrible burning. Induration of cellular tissue, abscess, bubo, and all inflammation of connective tissue in which there exists a purulent focus.
Tissues: Hemorrhages, black, thick, tar-like, rapidly decomposing, from any orifice. Glands swollen, cellular tissues edematous and indurated. Septicemia. Ulceration, sloughing and intolerable burning. Erysipelas. Black and blue blisters. Dissecting wounds. Insect stings. Bad effects from inhaling foul odors. Gangrenous parotitis. Succession of boils. Gangrene. Foul secretions.
Hydrastis Canadensis
Myalgic pain (chronic pain and fatigue) in scalp and muscles of neck. Eczema on forehead along line of hair. Sinusitis, after coryza. Eruption like variola. Lupus; ulcers, cancerous formations. General tendency to profuse perspiration and unhealthy skin.
Tends to blow nose all the time. Tongue white, swollen, large, flabby, slimy; shows imprint of teeth; as if scalded; stomatitis. Ulceration of tongue, fissures toward the edges. Follicular pharyngitis. Raw, smarting, excoriating sensation. Hawking of yellow, tenacious mucus. Goiter of puberty and pregnancy.
Rhus Toxicodendrone
Eyes swollen, red, edematous; orbital cellulitis. Pustular inflammations. Photophobia; profuse flow of yellow pus. Edema of lids, suppurative iritis. Lids inflamed, agglutinated swollen. Circumscribed corneal injection. Intensive ulceration of the cornea. Iritis, after exposure to cold and dampness, and of rheumatic origin. Eye painful on turning it or pressing. Skin red, swollen; itching intense. Vesicles, herpes; urticaria; pemphigus; erysipelas; vesicular suppurative forms. Glands swollen. Cellulitis. Burning eczematous eruptions with tendency to scale formation.
Tip of nose red, sore, ulcerated. Swelling of nose. Nosebleed on stooping. Jaws crack when chewing. Easy dislocation of jaw. Swollen face, erysipelas. Cheek bones sensitive to touch. Parotitis. Facial neuralgia, with chilliness; worse, evening.
Copaiva Officianalis
Hives, with fever and constipation. Roseola. Erysipelatous inflammation, especially around abdomen. Circumscribed lenticular patches, with itching; mottled appearance. Chronic urticaria in children. Bullous eruptions.
Phosphorus
Phosphorus should be advised when there is pain in ear and marked tinnitus/noises in ear, throbbing pain, tearing or shooting type. Irritates, inflammation and degeneration of mucous membranes, of serous membranes, inflammation of spinal cord and nerves, paralysis. Muscular pseudo-hypertrophy, neuritis. Inflammation of the respiratory tract. Edema of lids, eyes. Swelled and easily bleeding gums.
Conium Maculatum
Tearing and shooting in the ears, and round the ears. Accumulation of cerumen, which resembles moldy paper, and which is mixed with purulent mucus. Blood-colored cerumen. Roaring and humming in both ears, buzzing, tinkling, and rumbling in the ears. Painful sensibility of hearing. Diminution of hearing, ceasing when the cerumen is removed, and until it is renewed. Swelling and induration of the parotids.
Belladonna
Belladonna is an effective medicine to reduce inflammation of mastoid bone and relieve pain. Pain that can be throbbing, tearing, stitching or shooting type. The ear is also hot and sensitive to touch. It has a marked action on the vascular system, skin and glands. Belladonna always is associated with hot, red skin, flushed face, glaring eyes, throbbing carotids, excited mental state, hyperesthesia of all senses, delirium, restless sleep, convulsive movements, dryness of mouth and throat with aversion to water, neuralgic pains that come and go suddenly.
Face red, bluish-red, hot, swollen, shining; convulsive motion of facial muscles. Swelling of upper lip. Facial neuralgia with twitching muscles and flushed face. Eyes feel swollen and protruding, staring, brilliant; conjunctiva red; dry, burn; photophobia; shooting in eyes. Exophthalmos. Diplopia, squinting, spasms of lids. Eyelids swollen. Fundus congested.
Membrane tympani bulges and injected. Parotid gland swollen. Sensitive to loud tones. Hearing very acute. Otitis media. Pain causes delirium. Red and swollen nose. Bleeding of nose, with red face. Coryza; mucus mixed with blood.
Mouth dry, tongue red on edges. Strawberry tongue. Grinding of teeth. Tongue swollen and painful. Stammering. Throat dry, as if glazed; angry-looking congestion; red. Tonsils enlarged; throat feels constricted; difficult deglutition; worse, liquids. Sensation of a lump. Esophagus dry; feels contracted. Spasms in throat. Continual inclination to swallow. Scraping sensation. Muscles of deglutition very sensitive. Hypertrophy of mucous membrane.
Carbo Animalis
Carbo Animalis for sharp, stitching pain and swelling behind the ear. Feeble circulation and lowered vitality. Glands are indurated, veins distended, skin blue. Spongy ulcers, copper-colored eruption. Acne rosacea. Chilblains. Verruca on hands and face of old people, with bluish color of extremities. Glands indurated, swollen, painful, in neck, axillae, groin, breasts; pains lancinating, cutting, burning. Burning, rawness and fissures; moisture. Bubo.
Kali Muriaticum
It is a top listed medicine to resolve middle ear infection well as well as reduce the pain of mastoiditis. Catarrhal affections, in sub-acute inflammatory states, fibrinous exudations, and glandular swellings. White or gray coating of base of tongue, and expectoration of thick, white phlegm. White mucus in eyes, purulent scabs. Superficial ulcer. Trachoma. Corneal opacities.
Chronic, catarrhal conditions of the middle ear. Glands about the ear swollen. Snapping and noises in the ear. Threatened mastoid. Great effusion about the auricle. Cheek swollen and painful.
Aphthae; thrush; white ulcers in mouth. Swollen glands about jaw and neck. Coating of tongue grayish-white, dryish, or slimy. Follicular tonsillitis. Tonsils inflamed; enlarged so much, can hardly breathe. Grayish patches or spots in the throat and tonsils. Adherent crusts in vault of pharynx. “Hospital” sore throat. Eustachian catarrh.
Silicea Tera
It should be advised when there is pus in ear either from infection or mastoiditis. Swelling in the glabella. Swelling of lachrymal duct. Aversion to light. Stys. Iritis and irido-choroiditis, with pus in anterior chamber. Perforating or sloughing ulcer of cornea. Abscess in cornea after traumatic injury. Caries of mastoid. Dry, hard crusts form, bleeding when loosened. Nasal bones sensitive. Sneezing in morning. Obstructed and loss of smell. Perforation of septum.
Skin cracked on margin of lips. Eruption on chin. Facial neuralgia, throbbing, tearing, face red. Pricking as of a pin in tonsil. Colds settle in throat. Parotid glands swollen. Stinging pain on swallowing. Hard, cold swelling of cervical glands. Painless swelling of glands.
Aurum Metallicum
Inflammation of mastoid bone or may also have even started to decay. Mononucleosis, mastoiditis or kissing disease. Vascular cornea. Pains from without inward. Sticking pains inward. Trachoma with pannus. Ossicular caries and of mastoid. Obstinate fetid otorrhea after scarlatina. External meatus bathed in pus. Chronic nerve deafness; Labyrinthine disease due to syphilis.
Nose ulcerated, painful, swollen, obstructed. Inflammation of nose; caries; fetid discharge, purulent, bloody. Boring pains in nose; worse at night. Putrid smell from nose. Sensitive smell. Knobby tip of nose. Ulceration of gums. Tearing in zygoma. Mastoid and other facial bones inflamed. Stitches when swallowing; pain in glands. Caries of the palate.
Tellurium
Itching of hands and feet. Herpetic spots; ringworm. Ring-shape lesions, offensive odors from affected parts. Barber’s itch. Stinging in skin. Fetid exhalations. Offensive foot-sweat. Eczema, back of ears and occiput. Circular patches of eczema. Lids thickened, inflamed, itching. Pterygium; pustular conjunctivitis. Eczema. Itching, swelling, throbbing in meatus. Deafness. Nose obstructed; hawks salty phlegm from posterior nares.
Tuberculinium
Enlarged tonsils. Hard, dry cough during sleep. Shortness of breath. Sensation of suffocation. Hard, hacking cough, profuse sweating and loss of weight, rales all over chest. Chronic eczema; itching intense; worse at night. Acne in tuberculous patients. Measles; psoriasis.
Ferrum Phosphoricum
Ferrum Phos is a valuable medicine to manage cases in which soreness and swelling behind ear is marked. Throbbing earache. Mononucleosis, mastoiditis or kissing disease. Tinnitus. Eyes red, inflamed, with burning sensation, feeling as of sand under lids. Hyperemia of optic disc and retina, with blurred vision.
Face flushed; cheeks sore and hot. Florid complexion. Facial neuralgia; worse, shaking head and stooping. Throat and mouth hot; faucets red, inflamed. Ulcerated sore throat. Tonsils red and swollen. Eustachian tubes inflamed. Sore throat of singers. Diphtheria. Ranula in vascular, sanguine constitutions.
Capsicum
Constriction. Inflammation of petrous bone. Burning pains and general chilliness.. Mononucleosis, mastoiditis or kissing disease. Swelling and pain behind ears. Inflammation of mastoid. Tenderness over the petrous bone; extremely sore and tender to touch. Otorrhea and mastoid disease before suppuration.
Subacute inflammation of Eustachian tube with great pain – extending to the ears. Sore throat of smokers and drinkers. Smarting in; constriction. Burning constriction worse between acts of deglutition. Inflamed uvula and palate; swollen and relaxed. Herpes labialis. Stomatitis.
Magnesia Phosphorica
The great anti-spasmodic remedy. Cramping of muscles with radiating pains. Neuralgic pains. Mononucleosis, mastoiditis or kissing disease. Twitching of lids. Nystagmus strabismus, ptosis. Eyes hot, tired, vision blurred, colored lights before eyes. Ears – severe neuralgic pain; worse behind ears. Ulceration of teeth, with swelling of glands of face, throat and neck and swelling of tongue. Complaints of teething children. Spasms without febrile symptoms. Throat soreness and stiffness, inflamed, with chilliness, and aching all over.
I (Dr. Qaisar Ahmed) always prescribed highly diluted combination – TDs.
Complications
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Critical airway occlusion resulting in death,
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Acute laryngeal, pharynx and tongue swelling,
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Death from asphyxiation,
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Hereditary angioedema associated pancreatitis,
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Physicians should be mindful of cardiovascular instability including bradycardia after recombinant tissue plasminogen inhibitor in patients who take angiotensin-converting enzyme inhibitors.
P. S: This article is only for doctors and students having good knowledge about Homeopathy and allopathy.
For proper consultation and treatment, please visit our clinic.
None of above-mentioned medicine(s) is/are the full/complete treatment but just hints for treatment; every patient has his/her own constitutional medicine.
Dr. Sayyad Qaisar Ahmed (MD {Ukraine}, DHMS), Abdominal Surgeries, Oncological surgeries, Gastroenterologist, Specialist Homeopathic Medicines.
Senior research officer at Dnepropetrovsk state medical academy Ukraine.
Location: Al-Haytham clinic, Umer Farooq Chowk Risalpur Sadder (0923631023, 03119884588), K.P.K, Pakistan.
Find more about Dr Sayed Qaisar Ahmed at:
https://www.youtube.com/Dr Qaisar Ahmed