Alzheimer’s disease (AD) is a slowly progressive disease of the brain that is characterized by impairment of memory and eventually by disturbances in reasoning, planning, language, and perception.
It is possible that Alzheimer’s disease results from an increase in the production or accumulation of a specific protein (beta-amyloid protein) in the brain that leads to nerve cell death.
The likelihood of having Alzheimer’s disease increases substantially after the age of 70 and may affect 18% of population over the age of 85.
Mutations associated with Alzheimer’s disease have been found in about half of the patients with early-onset disease. In all of these patients, the mutation leads to excess production in the brain of a specific form of a small protein fragment called ABeta (Aβ). Many scientists believe that in the majority of sporadic (for example, non-inherited) cases of Alzheimer’s disease (these make up the vast majority of all cases of Alzheimer’s disease) there is too little removal of this Aβ protein rather than too much production.
Difference between Alzheimer’s disease and dementia
Dementia is a syndrome characterized by:
1.Impairment in memory,
2.Impairment in another area of thinking such as the ability to organize thoughts and reason, the ability to use language, or the ability to see accurately the visual world (not because of eye disease).
3.These impairments are severe enough to cause a decline in the patient’s usual level of functioning.
Although some kinds of memory loss are normal parts of aging, the changes due to aging are not severe enough to interfere with the level of function.
Many different diseases can cause dementia for example: hydrocephalus dementia, alcoholic dementia, Huntington’s dementia, trauma dementia etc. Alzheimer’s disease is the most common cause for dementia.
Risk factors
Age
The main risk factor for Alzheimer’s disease is increased age. As a population ages, the frequency of Alzheimer’s disease continues to increase. Fifteen percent of people over 65 years of age and 50% of those over 85 years of age have Alzheimer’s disease.
Genetics
There are also genetic risk factors for Alzheimer’s disease. Most people develop Alzheimer’s disease after age 70. However, less than 10% of people develop the disease in the fourth or fifth decade of life (40s or 50s). At least half of these early onset patients have inherited gene mutations associated with their Alzheimer’s disease.
The children of a patient with early onset Alzheimer’s disease who has one of these gene mutations has a 50% risk of developing Alzheimer’s disease.
Common forms of certain genes increase the risk of developing Alzheimer’s disease, but do not invariably cause Alzheimer’s disease. The best-studied “risk” gene is the one that encodes apolipoprotein E (apoE).
- The apoE gene has three different forms (alleles) — apoE2, apoE3, and apoE4.
- The apoE4 form of the gene has been associated with increased risk of Alzheimer’s disease in most (but not all) populations studied.
- The frequency of the apoE4 version of the gene in the general population varies but is always less than 30% and frequently 8% to 14%.
- People with one copy of the E4 gene usually have about a two- to three-fold increased risk of developing Alzheimer’s disease.
- Persons with two copies of the E4 gene (usually around 1% of the population) have about a nine-fold increase in risk.
- Nonetheless, even persons with two copies of the E4 gene don’t always get Alzheimer’s disease.
- At least one copy of the E4 gene is found in 40% of patients with sporadic or late-onset Alzheimer’s disease.
This means that in majority of patients with Alzheimer’s disease, no genetic risk factor has yet been found. Most experts do not recommend that adult children of patients with Alzheimer’s disease should have genetic testing for the apoE4 gene since there is no treatment for Alzheimer’s disease.
Estrogen
Many studies have found that women have a higher risk for Alzheimer’s disease than men. It is certainly true that women live longer than men, but age alone does not seem to explain the increased frequency in women.
The apparent increased frequency of Alzheimer’s disease in women has led to considerable research about the role of estrogen in Alzheimer’s disease. Recent studies suggest that allopathic estrogen should not be prescribed to post-menopausal women for the purpose of decreasing the risk of Alzheimer’s disease.
Nonetheless, the role of allopathic estrogen in Alzheimer’s disease remains an area of research focus. However, in Homeopathy, there are many proven medicines for Alzheimer’s disease.
Other risk factors for Alzheimer’s disease include: 
- High blood pressure (hypertension).
- Heart disease.
- Diabetes.
- Possibly elevated blood cholesterol.
- A majority of people with Down syndrome will develop the brain changes of Alzheimer’s disease by 40 years of age. This fact was also a clue to the “amyloid hypothesis of Alzheimer’s disease”.
- Some studies have found that Alzheimer’s disease occurs more often among people who suffered significant traumatic head injuries earlier in life, particularly among those with the apoE4 gene.
In the majority of Alzheimer’s disease cases, however, no specific genetic risks have yet been identified.
Warning signs and symptoms:
The following list of warning signs include common symptoms of Alzheimer’s disease:
1.Memory loss (forgetting important dates or events).
2.Difficulty performing familiar tasks (problems remembering the rules to a favorite game or driving to a familiar place).
3.Problems talking with others or writing (For example, a person may struggle to find the right words for items or names of people or places).
4.Disorientation to time and place (for example, forgetting where they are, losing track of the seasons, dates, and passage of time).
5.Poor or decreased judgment (for example, poor hygiene or poor judgment when dealing with money or financial matters).
6.Vision problems (problems reading or judging distances).
7.Problems with solving problems or planning (for example, problems tracking regular bills or following familiar recipes).
8.Misplacing things (for example, a person put items in unusual places and then are not able to retrace their steps find them again).
9.Changes in mood, personality, or behavior.
10.Loss of initiative or withdrawal from social or work activities.
It is normal for certain kinds of memory, such as the ability to remember lists of words, to decline with normal aging.
Normal individuals 50 years of age will recall only about 60% as many items on some kinds of memory tests as individuals 20 years of age. Furthermore, everyone forgets, and every 20 year old is well aware of multiple times he or she couldn’t think of an answer on a test that he or she once knew. Almost no 20 year old worries when he/she forgets something, that he/she has the ‘early stages of Alzheimer’s disease,’ whereas an individual 50 or 60 years of age with a few memory lapses may worry that they have the ‘early stages of Alzheimer’s disease.
Stages
The onset of Alzheimer’s disease is usually gradual, and it is slowly progressive. Memory problems that family members initially dismiss as “a normal part of aging” are in retrospect noted by the family to be the first stages of Alzheimer’s disease.
Problems of memory, particularly for recent events (short-term memory) are common early in the course of Alzheimer’s disease. For example, the individual may, on repeated occasions, forget to turn off an iron or fail to recall which of the morning’s medicines were taken. Mild personality changes, such as less spontaneity, apathy, and a tendency to withdraw from social interactions, may occur early in the illness.
As the disease progresses, problems in abstract thinking and in other intellectual functions develop. The person may begin to have trouble with figures when working on bills, with understanding what is being read, or with organizing the day’s work. Further disturbances in behavior and appearance may also be seen at this point, such as agitation, irritability, quarrelsomeness, and a diminishing ability to dress appropriately.
Later in the course of the disorder, affected individuals may become confused or disoriented about what month or year it is, be unable to describe accurately where they live, or be unable to name a place being visited. Eventually, patients may wander, be unable to engage in conversation, erratic in mood, uncooperative, and lose bladder and bowel control. In late stages of the disease, persons may become totally incapable of caring for themselves.
Diagnosis
No specific blood test or imaging test exists for the diagnosis of Alzheimer’s disease. Alzheimer’s disease is diagnosed when:
1.A person has sufficient cognitive decline to meet criteria for dementia;
2.The clinical course is consistent with that of Alzheimer’s disease;
3.No other brain diseases or other processes are better explanations for the dementia.
What other conditions should be screened for besides Alzheimer’s disease?
Ten other causes of dementia are:
Neurological disorders:
Parkinson’s disease, cerebrovascular disease and strokes, brain tumors, blood clots, and multiple sclerosis can sometimes be associated with dementia although many patients with these conditions are cognitively normal.
Infectious diseases:
Some brain infections such as chronic syphilis, chronic HIV, or chronic fungal meningitis can cause dementia.
Side effects of medications:
Many allopathic medicines can cause cognitive impairment, especially in elderly patients. Perhaps the most frequent offenders are allopathic drugs used to control bladder urgency and incontinence. “Psychiatric medications” such as antidepressants and anti-anxiety medications and “neurological medications” such as anti-seizure medications can also be associated with cognitive impairment.
If a physician evaluates a person with cognitive impairment who is on one of these medications, the medication is often gently tapered and/or discontinued to determine whether it might be the cause of the cognitive impairment. If it is clear that the cognitive impairment preceded the use of these medications, such tapering may not be necessary. On the other hand, “psychiatric,” “neurological,” and “incontinence” medications are often appropriately prescribed to patients with Alzheimer’s disease. Such patients need to be followed carefully to determine whether these medications cause any worsening of cognition.
Psychiatric disorders:
In older persons, some forms of depression can cause problems with memory and concentration that initially may be indistinguishable from the early symptoms of Alzheimer’s disease. Sometimes, these conditions, referred to as pseudodementia, can be reversed. Studies have shown that persons with depression and coexistent cognitive (thinking, memory) impairment are highly likely to have an underlying dementia when followed for several years.
Substance Abuse:
Abuse of drugs and alcohol abuse is often associated with cognitive impairment.
Metabolic Disorders:
Thyroid dysfunction, some steroid disorders, and nutritional deficiencies such as vitamin B12 deficiency or thiamine deficiency are sometimes associated with cognitive impairment.
Trauma:
Significant head injuries with brain contusions may cause dementia. Blood clots around the outside of the brain (subdural hematomas) may also be associated with dementia.
Toxic Factors:
Long term consequences of acute carbon monoxide poisoning can lead to an encephalopathy with dementia. In some rare cases, heavy metal poisoning can be associated with dementia.
Tumors:
Many primary and metastatic brain tumors can cause dementia. However, many patients with brain tumors have no or little cognitive impairment associated with the tumor.
The importance of comprehensive clinical evaluation
Because many other disorders can be confused with Alzheimer’s disease, a comprehensive clinical evaluation is essential in arriving at a correct diagnosis.
Such an assessment should include at least three major components:
1) Thorough general medical workup.
2) Neurological examination including testing of memory and other functions of thinking.
3) A psychiatric evaluation to assess mood, anxiety, and clarity of thought.
Include imaging of the brain in the initial evaluation of patients with dementia, said Dr Qaisar Ahmed neither a non-contrast CT scan or an MRI scan. Other imaging procedures that look at the function of the brain (functional neuroimaging), such as SPECT, PET, and MRI, may be helpful in specific cases, but generally are not needed.
Despite many attempts, identification of a blood test to diagnose Alzheimer’s disease has remained elusive. Such testing is neither widely available nor recommended.
Allopathic treatment and management for Alzheimer’s disease
The management of Alzheimer’s disease consists of medication based and non-medication-based treatments. Two different classes of pharmaceuticals for treating Alzheimer’s disease: cholinesterase inhibitors and partial glutamate antagonists.
Neither class of drugs has been proven to slow the rate of progression of Alzheimer’s disease.
Nonetheless, many clinical trials suggest that Homeopathic medications are superior to allopathic in treating Alzheimer’s disease.
Alzheimer’s disease medications
Cholinesterase inhibitors (ChEIs):
In patients with Alzheimer’s disease there is a relative lack of a brain chemical neurotransmitter called acetylcholine. (Neurotransmitters are chemical messengers produced by nerves that the nerves use to communicate with each other in order to carry out their functions.) Substantial research has demonstrated that acetylcholine is important in the ability to form new memories. The cholinesterase inhibitors (ChEIs) block the breakdown of acetylcholine. As a result, more acetylcholine is available in the brain, and it may become easier to form new memories.
Donepezil hydrochloride, Rivastigmine, and Galantamine and Tacrine are used by most physicians. Most experts in Alzheimer’s disease do not believe there is an important difference in the effectiveness of these three allopathic drugs.
Several studies suggest that the progression of symptoms of patients on these drugs seems to plateau for six to 12 months, but inevitably progression then begins again.
It is not known whether rivastigmine and galantamine are also effective in severe Alzheimer’s disease.
The principal side effects of ChEIs involve the gastrointestinal system and include nausea, vomiting, cramping, diarrhea, happatic and spleen diseases, renal failure, stroke, sudden death etc.
Partial glutamate antagonists:
Glutamate is the major excitatory neurotransmitter in the brain. One theory suggests that too much glutamate may be bad for the brain and cause deterioration of nerve cells.
Memantine works by partially decreasing the effect of glutamate to activate nerve cells. Memantine is approved for treatment of moderate and severe dementia, but studies did not show it was helpful in dementia.
Other medications for Alzheimer’s disease:
Namzaric is approved for use as a fixed-dose combination of memantine hydrochloride extended-release (an NMDA receptor antagonist) and donepezil hydrochloride (an acetylcholinesterase inhibitor) for treatment of moderate to severe Alzheimer’s, but with care because of their side effects on liver, spleen, gallbladder and kidneys.
Treatment of psychiatric symptoms in Alzheimer’s disease
Symptoms of Alzheimer’s disease include agitation, depression, hallucinations, anxiety, and sleep disorders.
Standard psychiatric drugs are widely used to treat these symptoms although none of these drugs have been specifically approved for treating these symptoms in patients with Alzheimer’s disease.
Nevertheless, frequently these symptoms are so severe that it becomes impossible for caregivers to take care of the patient, and treatment with medication to control these symptoms becomes necessary. Agitation is common, particularly in middle and later stages of Alzheimer’s disease.
Many different classes of allopathic drugs have been tried to treat agitation including:
- Antipsychotics,
- Mood-stabilizing anticonvulsants,
- Trazodone,
- Anxiolytics, and
- Beta-blockers.
Studies are conflicting about the usefulness of these different drug classes. It was thought that newer, atypical antipsychotic agents such as clozapine, risperidone, olanzapine, quetiapine, and ziprasidone might have advantages over the older antipsychotic agents because of their fewer and less severe side effects and the patient’s ability to tolerate them.
However, more recent studies have not demonstrated superiority of the newer antipsychotics. Some research shows that these newer antipsychotics may be associated with increased risk of stroke or sudden death than the older antipsychotics.
Apathy and difficulty concentrating occur in most Alzheimer’s disease patients and should not be treated with antidepressant drugs. However, many Alzheimer’s disease patients have other symptoms of depression including sustained feelings of unhappiness and/or inability to enjoy their usual activities.
Such patients may advise a trial of antidepressant drugs. Most physicians will try selective serotonin reuptake inhibitors (SSRIs), such as sertraline, citalopram, or fluoxetine, as first-line drugs for treating depression in Alzheimer’s disease.
Anxiety is another symptom in Alzheimer’s disease that occasionally requires treatment. Benzodiazepines such as diazepam or lorazepam may be associated with increased confusion and memory impairment. Non-benzodiazepine anxiolytics, such as buspirone or SSRIs, are probably preferable.
Difficulty sleeping (insomnia) occurs in many patients with Alzheimer’s disease at some point in the course of their disease. Many Alzheimer’s disease specialists prefer the use of sedating atypical antidepressants such as trazodone.
To date, no treatment which can reverse the process of Alzheimer’s disease, delirium, dementia etc, has been identified.
Homeopathic treatment for Alzheimer’s disease
Anacardium Orientale
Neurasthenics patients; who have nervous dyspepsia, relieved by food; impaired memory, depression, and irritability; diminution of senses (smell, sight, hearing). Fears. Weakening of all senses, sight, hearing, etc. Aversion to work; lacks self-confidence; irresistible desire to swear and curse. Sensation of a plug in various parts-eyes, rectum, bladder, etc; also of a band. Empty feeling in stomach; eating temporarily relieves all discomfort. Alzheimer’s disease. Dementia. Delirium. Intermittency of symptoms.
Fixed ideas. Hallucinations: thinks he is possessed of two persons or wills. Anxiety when walking, as if pursued. Profound melancholy and hypochondriasis, with tendency to use violent language. Brain-fag. Impaired memory. Absent mindedness. Very easily offended. Malicious; seems bent on wickedness. Lack of confidence in himself or others. Suspicious. Clairaudient hears voices far away or of the dead. Senile dementia. Absence of all moral restraint. Vertigo. Pressing pain, as from a plug; worse after mental exertion-in forehead; occiput, temples, vertex; better during a meal. Itching and little boils on scalp.
Hyoscymaus Niger
Disturbed nervous system. It is as if some diabolical force took possession of the brain and prevented its functions. Mania of a quarrelsome and obscene character. Inclined to be unseemly and immodest in acts, gestures and expressions. Very talkative, and persists in stripping herself, or uncovering genitals. Is jealous, afraid of being poisoned, etc. Its symptoms also point to weakness and nervous agitation – Alzheimer’s disease. Tremulous weakness and twitching of tendons. Subsultus tendinum. Muscular twitching, spasmodic affections, generally with delirium. Non-inflammatory cerebral activity.
Toxic gastritis. Very suspicious. Obscene, lascivious mania uncovers body; jealous, foolish. Great hilarity; inclined to laugh at everything. Delirium, with attempt to run away. Low, muttering speech; constant carphologia, deep stupor.
Head feels light and confused. Vertigo as if intoxicated. Brain feels loose, fluctuating. Inflammation of brain, with unconsciousness; head is shaken to and fro. Alzheimer’s disease.
Alumina
Low spirited; fears loss of reason. Confused as to personal identity. Hasty, hurried. Time passes slowly. Variable mood. Better as day advances. Suicidal tendency when seeing knife or blood. Alzheimer’s disease. Stitching burning pain in head, with vertigo relieved by food. Pressure in forehead. Inability to walk closed eyes. Throbbing headache, with constipation. Vertigo, with nausea; better after breakfast. Falling out of hair; scalp itches and is numb.
Rauwolfia Serpentina
Melancholia include are Abasement, Abuser, Abjection, Abjectness, Bleakness, Bummer, Cheerlessness, Dejection, Desolation, Desperation, Despondency, Disconsolation, Discouragement, Dispiritedness, Distress, Dole, Dolor, Dreariness, Dullness, Dumps, Ennui, Gloom, Gloominess, Hopelessness, Lowness, Melancholy, Misery, Mortification, Qualm, Sadness, Sorrow, Trouble, Unhappiness, Vapors, Woefulness, Worry, Downheartedness, Dolefulness, Blue Funk, Blahs, Heaviness Of Heart and Lugubrious. Paranoia, Paranoia. Alzheimer’s disease.
Lac Caninum
Very forgetful; in writing, makes mistakes. Despondent; thinks her disease incurable. Attacks of rage. Visions of snakes. Thinks himself of little consequence. Alzheimer’s disease. Sensation of walking or floating in the air. Pain first one side, then the other. Blurred vision, nausea and vomiting at height of attack of headache. Occipital pain, with shooting extending to forehead. Sensation as if brain were alternately contracted and relaxed. Noises in ears. Reverberation of voice.
Medorrhinum
Severe disturbance and irritability of nervous system. Dwarfed and stunted. State of collapse and trembling all over. History of sycosis. Alzheimer’s disease. Intensity of all sensations. Weak memory. Loses the thread of conversation. Cannot speak without weeping. Time passes too slowly. Is in a great hurry. Hopeless of recovery. Difficult concentration. Fears going insane. Sensibility exalted. Nervous, restless. Fear in the dark and of some one behind her. Melancholy, with suicidal thoughts. Burning pain in brain; worse, occiput. Head heavy and drawn backward. Headache from jarring of cars, exhaustion, or hard work. Weight and pressure in vertex.
Macinela
Silent mood, sadness. Wandering thoughts. Sudden vanishing of thought. Bashful. Fear of becoming insane. Vertigo; head feels lights, empty. Scalp itches. Hair falls out after acute sickness. Fear: of getting crazy; of evil spirits. Alzheimer’s disease. Averse to work and answering questions. Sadness. Anxiety; before menses. Homesick. Bashful and taciturn; timid look.
Datura Metel
soporose condition, and later delirium and spasms. The soporose state may be absent. Delirium may be vociferous, or merely garrulous. Patient usually manifests excessive timidity. Picks at real or imaginary objects. Performs ridiculous antics. Several movements appear due to perverted vision, and inability to judge distances. After the delirium, patient remembers nothing of what has occurred. Extreme dilatation of pupils. Flickering before eyes with photophobia. Pulse and temperature undergo extremes of exaltation and depression. Alzheimer’s disease. Convulsions. Delirium. Epilepsy. Eye affections. Mania. Timidity.
Argentum Nitricum
The neurotic effects of Argentum Nitricum are very marked, many brain and spinal symptoms presenting, head symptoms often determine the choice of this remedy. Symptoms of incoordination, loss of control and want of balance everywhere, mentally and physically, trembling in affected parts. Alzheimer’s disease. Gastroenteritis. Great desire for sweets, the splinter-like pains, and free muco-purulent discharge in the inflamed and ulcerated mucous membranes. Sensation as if a part were expanding and other errors of perception are characteristic.
Withered up and dried constitutions present a favorable field for its action, especially when associated with unusual or long continued mental exertion. Pains increase and decrease gradually. Flatulent state and prematurely aged look. Explosive belching especially in neurotics. Upper abdominal infections brought on by undue mental exertion. Paraplegia Myelitis and disseminated sclerosis of brain and cord. Intolerance of heat. Sensation of a sudden pinch. Destroyed red blood corpuscles – anemia.
Thinks his understanding will and must fail. Fearful and nervous; impulse to jump out of window. Faintish and tremulous. Melancholic; apprehensive of serious disease. Time passes slowly. Memory weak. Errors of perception. Impulsive; wants to do things in a hurry. Peculiar mental impulses. Fears and anxieties and hidden irrational motives for actions.
Headache with coldness and trembling. Emotional disturbances cause appearance of hemi cranial attacks. Sense of expansion. Brain-fag, with general debility and trembling. Headache from mental exertion, from dancing. Vertigo, with buzzing in ears and with nervous affections. Aching in frontal eminence, with enlarged feeling in corresponding eye. Boring pain; better on tight bandaging and pressure. Itching of scalp. Hemicrania; bones of head feel as if separated.
Cannabis Indica
A condition of intense exaltation, in which all perceptions and conceptions, all sensations and all emotions are exaggerated to the utmost degree.
Subconscious or dual nature state; Dual personality disorder. Apparently under the control of the second self, but, the original self, prevents the performance of acts which are under the domination of the second self. Apparently the two natures cannot act independently, one acting as a check, upon the other.
Most remarkable hallucinations and imaginations, exaggeration of the duration of time and extent of space. Conception of time, space and place is gone. Extremely happy and contented, nothing troubles. Ideas crowd upon each other. Epilepsy, mania, dementia, delirium tremens, and irritable reflexes. Exophthalmic goiter. Catalepsy. Alzheimer’s disease.
Excessive loquacity; exuberance of spirits. Constantly theorizing. Anxious depression; constant fear of becoming insane. Mania, must constantly move. Very forgetful; cannot finish sentence. Is lost in delicious thought. Uncontrollable laughter. Delirium tremens. Clairvoyance. Emotional excitement; rapid change of mood. Cannot realize her identity, chronic vertigo as of floating off. Feels as if top of head were opening and shutting and as if calvarium were being lifted. Shocks through brain. Uraemic headache with flatulence. Involuntary shaking of head. Migraine attack preceded by unusual excitement with loquacity.
Ignatia Amara
Hyperesthesia of all senses. Tendency to colonic spasms. Mentally, the emotional element is uppermost, and co-ordination of function is interfered with. It is one of the chief remedies for hysteria. Nervous temperament-women of sensitive, easily excited nature, dark, mild disposition, quick to perceive, rapid in execution. Rapid change of mental and physical condition, opposite to each other. Alzheimer’s disease. Alert, nervous, apprehensive, rigid, trembling patients who suffer acutely in mind or body, at the same time. Effects of grief and worry. Cannot bear tobacco. Pain is small, circumscribed spots. The plague. Hiccough and hysterical vomiting.
Changeable mood; introspective; silently brooding. Melancholic, sad, tearful. Not communicative. Sighing and sobbing. Aftershocks, grief, disappointment. Head feels hollow, heavy; worse, stooping. Headache as if a nail were driven out through the side. Cramp-like pain over root of nose. Congestive headaches following anger or grief; worse, smoking or smelling tobacco, inclines head forward.
Coca
Melancholy. Hypochondriasis. Mental depression with drowsiness. Bashfulness. Prefers solitude and darkness. Alzheimer’s disease. Muddled feeling in brain. Loss of energy. Great mental excitement. Vertigo and fainting. Tension over forehead. Headache just over eyebrows. Shocks in head; dull, full feeling in occiput with vertigo, the only possible position is on the face.
Kali Phosphoricum
One of the greatest nerve medicines. Prostration. Weak and tired. Marked disturbance of the sympathetic nervous system. Conditions arising from want of nerve power, neurasthenia, mental and physical depression. Alzheimer’s disease. The causes are usually excitement, overwork and worry. Adynamia and decay, gangrenous conditions. Suspected malignant tumors. After removal of cancer when in healing process skin is drawn tight over the wound. Delayed labor.
Anxiety, nervous dread, lethargy. Indisposition to meet people. Extreme lassitude and depression. Very nervous, starts easily, irritable. Brain-fag; hysteria; night terrors. Somnambulance. Loss of memory. Slightest labor seems a heavy task. Great despondency about business. Shyness; disinclined to converse.
Occipital headache. Vertigo, from lying, on standing up, from sitting, and when looking upward. Cerebral anaemia. Headache of students, and those worn out by fatigue. Headaches are relieved by gentle motion. Headache, with weary, empty, gone feeling at stomach.
Aurum Metallicum
Feeling of self-condemnation and utter worthlessness. Profound despondency, with increased blood pressure, with thorough disgust of life, and thoughts of suicide. Talks of committing suicide. Fear of death. Peevish and vehement at least contradiction. Alzheimer’s disease. Anthropophobia. Mental derangements. Constant rapid questioning without waiting for reply. Cannot do things fast enough. Over sensitiveness; to noise, excitement, confusion.
Violent pain in head; worse at night, outward pressure. Roaring in head. Vertigo. Tearing through brain to forehead. Pain in bones extending to face. Congestion to head. Boils on scalp.
Nux Moschata
Tendency to fainting fits, with heart failure. Cold extremities, extreme dryness of mucous membranes and skin. Strange feeling, with irresistible drowsiness. Indicanuria. General inclination to become unconscious during acute attacks. Alzheimer’s disease. Lypothymia. Staggers on trying to walk.
Mind changeable; laughing and crying. Confused, impaired memory. Bewildered sense, as in a dream. Thinks she has two heads. Vertigo when walking in open air; aches from eating a little too much. Feeling of expansion, with sleepiness. Pulsating in head. Cracking sensation in head. Sensitive to slightest touch in a draught of air. Bursting headache; better hard pressure.
Prognosis for Alzheimer’s disease
Alzheimer’s disease is invariably progressive over two to 25 years with most patients in the eight-to-15-year range. Nonetheless, defining when Alzheimer’s disease starts, particularly in retrospect, can be very difficult.
With allopathic treatment, a person with Alzheimer’s disease may no longer be able to do math but still may be able to read a magazine with pleasure. Playing the piano might become too stressful in the face of increasing mistakes, but singing along with others may still be satisfying. The chessboard may have to be put away, but playing tennis may still be enjoyable. Challenge, frustration, closeness, anger, warmth, sadness, and satisfaction may all be experienced by those who work to help the person with Alzheimer’s disease.
The reaction of a patient with Alzheimer’s disease to the illness and his or her capacity to cope with it also vary and may depend on such factors as lifelong personality patterns and the nature and severity of stress in the immediate environment. Depression, severe uneasiness, paranoia, or delusions may accompany or result from the disease, but these conditions can often be improved by appropriate treatments.
Although in allopathy there is no cure for Alzheimer’s disease, treatments are available to alleviate many of the symptoms that cause suffering.
However, many clinical trials confirmed that with Homeopathic treatment, Alzheimer’s disease is curable, and patient could live almost normal life after two-three months of treatmen.
P. S: This article is only for doctors having good knowledge about Homeopathy and allopathy, for learning purpose(s).
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Senior research officer at Dnepropetrovsk state medical academy Ukraine.
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