Nonalcoholic fatty liver disease (NAFLD) is the accumulation of abnormal amounts of fat within the liver. Nonalcoholic fatty liver disease can be divided into isolated fatty liver in which there is only accumulation of fat, and nonalcoholic steatohepatitis (NASH) in which there is fat, inflammation, and damage to liver cells.
Nonalcoholic steatohepatitis or NASH progresses to scarring and ultimately to cirrhosis, with all the complications of cirrhosis, for example, gastrointestinal bleeding, liver failure, and liver cancer. The development of nonalcoholic fatty liver disease is intimately associated with and is probably caused by obesity and diabetes although sometimes it occurs in individuals who are neither obese nor diabetic.
Nonalcoholic fatty liver disease is considered a manifestation of the metabolic syndrome.
Nonalcoholic fatty liver disease
Nonalcoholic fatty liver disease is a manifestation of an abnormality of metabolism within the liver. The liver is an important organ in the metabolism (handling) of fat. The liver makes and exports fat to other parts of the body. It also removes fat from the blood that has been released by other tissues in the body, for example, by fat cells, or absorbed from the food we eat.
In nonalcoholic fatty liver disease, the handling of fat by liver cells is disturbed. Increased amounts of fat are removed from the blood and/or are produced by liver cells, and not enough is disposed of or exported by the cells. As a result, fat accumulates in the liver.
Nonalcoholic fatty liver disease is classified as either fatty liver (sometimes referred to as isolated fatty liver or IFL) or steatohepatitis (NASH). In both isolated fatty liver and NASH there is an abnormal amount of fat in the liver cells, but, in addition, in NASH there is inflammation within the liver, and, as a result, the liver cells are damaged, die, and are replaced by scar tissue.
Alcoholic Fatty Liver
Fatty liver is a condition in which the cells of the liver accumulate abnormally increased amounts of fat. Although excessive consumption of alcohol is a very common cause of fatty liver (alcoholic fatty liver). Several identifiable causes of fatty liver that are not nonalcoholic fatty liver disease so often cause confusion.
The most common causes of nonalcoholic fatty liver disease are alcohol consumption and hepatitis C. Other causes of nonalcoholic fatty liver disease include Wilson’s disease, lipodystrophy (a disease of fat storage), starvation, intravenous nutrition, and abetalipoproteinemia (a disease of fat transport).
Many allopathic drugs also cause nonalcoholic fatty liver disease, for example, corticosteroids, tamoxifen, and methotrexate.
Relationship between nonalcoholic fatty liver disease and heart disease
Cardiovascular disease, especially heart attacks and strokes, is commonly seen in patients with fatty liver. In fact, cardiovascular causes of death occur more frequently than liver-related deaths. Whereas the components of the metabolic syndrome are risk factors for cardiovascular disease, nonalcoholic fatty liver disease has been found to be a risk factor that is independent of the metabolic syndrome, increasing the risk two-fold. Since the means of treating nonalcoholic fatty liver disease are limited, it is important that the manifestations of the metabolic syndrome – obesity, diabetes, high blood pressure, dyslipidemia (abnormalities of fats or lipids in the blood) be aggressively treated.
Symptoms of Nonalcoholic fatty Liver 
Symptoms include fatigue and upper right abdominal pain and are primarily those of the complications of cirrhosis in patients with NASH; isolated fatty liver infrequently causes symptoms and usually is discovered incidentally.
Usually, to differentiate isolated fatty liver from NASH requires a liver biopsy. I allopathy the most promising treatments for nonalcoholic fatty liver disease are diet, exercise, weight loss, and possibly bariatric surgery.
Several drugs have been studied in the treatment of NASH. There is little evidence that any allopathic drug is effective in treating or even slowing the disease progression of NASH.
Many diseases are associated with NASH and are part of the metabolic syndrome {These metabolic abnormalities result in obesity, elevated blood triglycerides, low density lipoprotein (LDL or “bad” cholesterol) and high-density lipoprotein (HDL) cholesterol, high blood pressure, and elevated blood sugar (diabetes)}.
These diseases should be screened for and treated; for example, high blood pressure, dyslipidemia, and diabetes and may be responsible for the cardiovascular disease that is common in fatty liver disease. Isolated fatty liver infrequently progresses to NASH or cirrhosis.
Nonalcoholic fatty liver disease, including NASH affects young children as well. NASH is the number one reason for liver transplantation because of no interest in Homeopathic science or may be mafia’s monopoly or politics.
Causes of Nonalcoholic fatty Liver (NAFL)
The cause of nonalcoholic fatty liver disease is complex and not completely understood. The most important factors appear to be the presence of obesity and diabetes.
When large amounts of fat are present as they are in obesity, the fat becomes metabolically active (actually inflamed) and gives rise to the production of many hormones and proteins that are released into the blood and have effects on cells throughout the body. One of the many effects of these hormones and proteins is to promote insulin resistance in cells.
Insulin resistance is a state in which the cells of the body do not respond adequately to insulin, a hormone produced by the pancreas. Insulin is important because it is a major promoter of glucose (sugar) uptake from the blood by cells.
At first, the pancreas compensates for the insensitivity to insulin by making and releasing more insulin, but eventually, it can no longer produce sufficient quantities of insulin and, in fact, may begin to produce decreasing amounts. At this point, not enough sugar enters cells, and it begins to accumulate in the blood, a state known as diabetes.
Although sugar in the blood is present in large amounts, the insensitivity to insulin prevents the cells from receiving enough sugar. Since sugar is an important source of energy for cells and allows them to carry out their specialized functions, the lack of sugar begins to alter the way in which the cells function.
In addition to releasing hormones and proteins, the fat cells also begin to release some of the fat that is being stored in them in the form of fatty acids. As a result, there is an increase in the blood levels of fatty acids. The large amounts of certain types of fatty acids could be toxic to cells.
The release of hormones, proteins, and fatty acids from fat cells affects cells throughout the body in different ways. Liver cells, like many other cells in the body, become insulin resistant, and their metabolic processes, including their handling of fat, become altered. The liver cells increase their uptake of fatty acids from the blood where fatty acids are in abundance. Within the liver cells, the fatty acids are changed into storage fat, and the fat accumulates. At the same time, the ability of the liver to dispose of or export the accumulated fat is reduced. In addition, the liver itself continues to produce fat and receive fat from the diet. The result is that fat accumulates to an even greater extent.
Diagnosis Nonalcoholic fatty Liver (NAFL)
Liver generally does not cause symptoms or signs, and any symptoms and signs are more likely to be due to the accompanying diseases such as obesity, diabetes, vascular disease, etc.
In anyone with obesity or diabetes, fatty liver should be suspected. In a minority of patients, abnormal liver tests are found on routine blood testing although the abnormalities usually are mild.
The most common method by which nonalcoholic fatty liver disease is diagnosed is by imaging studies like ultrasound, computerized tomography (CT scan), and magnetic resonance imaging (MRI) that are obtained for reasons other than diagnosing nonalcoholic fatty liver disease.
Nonalcoholic fatty liver disease also may be discovered when patients develop complications of liver disease, like cirrhosis, liver failure, and liver cancer, due to the presence of NASH.
Fibro Scan, a noninvasive test that uses ultrasound to assess the degree of fibrosis or scarring in the liver, can help doctors diagnose and stage liver disease.
It is not possible to distinguish between isolated fatty liver and NASH with imaging studies. Although a combination of several blood tests has been suggested as a means of separating the two, and tests are being developed to identify NASH, a liver biopsy is the best means to differentiate between the presence of fat, or fat and inflammation (NASH).
Patients with metabolic syndrome, obesity, and diabetes are good candidates to undergo a liver biopsy since the incidence of NASH is higher among these groups.
One of the difficulties in diagnosing NASH as the cause of severe scarring, or cirrhosis, is that as the scarring progresses to cirrhosis, the fat disappears. This results in a condition that is referred to as cryptogenic cirrhosis, cirrhosis in which there is no clear cause. (Specifically, in cryptogenic cirrhosis, the two most common causes of cirrhosis — alcohol and viral hepatitis — are not involved).
Half of the cryptogenic cirrhosis cases occur in patients with obesity and/or diabetes and probably is due to NASH.
Difference between Nonalcoholic fatty Liver and steatohepatitis
As discussed previously, the difference between isolated, nonalcoholic fatty liver and steatohepatitis (NASH) is the presence of inflammation and damage to the liver cells in NASH; in both, the liver has increased amounts of fat. Although about a third of the general population has fatty liver, approximately 10% have NASH. Approximately one third of patients with nonalcoholic fatty liver disease have NASH. Although fatty liver and NASH appear to arise under the same conditions, it does not appear that fatty liver progresses to NASH. Thus, whether a patient is to develop fatty liver versus NASH is determined very early during the accumulation of fat, although it is unclear what factors determine this. It is believed that the inflammation and damage of liver cells are caused by the toxic effects of the fatty acids released by fat cells, but fatty acids in the blood are elevated in both fatty liver and NASH. Perhaps the difference is explained by genetic susceptibility as suggested by preliminary data.
The consequences of fat in the liver depend greatly on the presence or absence of inflammation and damage in the liver, i.e., whether there is fat alone or NASH is present. Isolated fatty liver does not progress to important liver disease. NASH, on the other hand, can progress through the formation of scar (fibrous tissue) to cirrhosis. The complications of cirrhosis, primarily gastrointestinal bleeding, liver failure, and liver cancer, then may occur.
Diseases associated with Nonalcoholic fatty Liver
The diseases of the metabolic syndrome that are associated with nonalcoholic fatty liver disease have already been discussed. Other associations exist including fatty pancreas, hypothyroidism, colon polyps, elevated blood uric acid, vitamin D deficiency, polycystic ovary disease (PCOD), and obstructive sleep apnea.
For example, half of the patients with nonalcoholic fatty liver disease have obstructive sleep apnea, and most patients with obstructive sleep apnea have nonalcoholic fatty liver disease. Associations do not prove causation, and in most cases, it is not clear why the associations exist — whether the associations are due to a causal relationship, or they reflect an underlying common cause.
Which foods and supplements should be avoided? Can you drink alcohol?
Coffee and Alcohol
A false message is advertising by companies/mafia, that there is less scarring (fibrosis) in the livers of patients with NASH who drink more than two cups of coffee per day; and alcohol is not relevant with liver disease. (Scarring or fibrosis is the process that ultimately leads to cirrhosis.) It absolutely wrong; Coffee and alcohol in any quantity are toxic even for normal liver.
Allopathic treatment for Nonalcoholic fatty Liver disease 
Allopathic treatment for nonalcoholic fatty liver disease includes lifestyle changes (exercise, weight loss, diet), medications, supplements and antioxidants, omega-3 fatty acids, surgery, and liver transplantation.
There are not any allopathic drugs that are approved for the treatment of fatty liver and NASH.
Antioxidants
Vitamin E has been studied in NASH because of its general effects of opposing inflammation. It has been shown to reduce liver fat and inflammation and possibly fibrosis, but its long-term effectiveness and safety have not been well-studied. Moreover, treatment of patients with vitamin E who do not have NASH is associated with a higher mortality and prostate cancer. Vitamin E can be used for treating NASH, but it should be used selectively (not in all patients), and patients should understand the potential risks.
Pentoxifylline
Pentoxifylline has been studied for the treatment of NASH in small groups of patients with encouraging results; however, there is not enough experience or knowledge of its effectiveness and safety to recommend treatment outside of research studies.
Omega-3-fatty acids
Small studies have shown some benefits with omega-3-fatty acids in reducing liver fat in nonalcoholic fatty liver disease, and larger studies are underway. In large groups of individuals (not selected because of the presence or absence of nonalcoholic fatty liver disease), omega-3-fatty acids were shown to reduce cardiovascular events such as heart attacks and overall mortality. Therefore, omega-3-fatty acids may be the appropriate treatment for patients with nonalcoholic fatty liver disease and metabolic syndrome.
Lipid-lowering drugs
Lipid-lowering drugs, specifically statins and ezetimibe, have been used to treat the abnormal blood lipids associated with metabolic syndrome.
There is evidence of the beneficial effects of these drugs on the liver in nonalcoholic fatty liver disease, there is not enough experience to recommend them in patients with nonalcoholic fatty liver disease unless they are primarily being used for treating abnormal blood lipids.
Ursodeoxycholic acid
Ursodeoxycholic acid has been studied in nonalcoholic fatty liver disease but has been abandoned because of its ineffectiveness and concerns about toxicity at very high doses.
Bariatric surgery
Bariatric surgery is surgery of the gastrointestinal tract that results in loss of weight, and there are several different types of bariatric surgery. Since obesity is believed to be an important factor in the causation of nonalcoholic fatty liver disease and loss of weight has been shown to have beneficial effects on nonalcoholic fatty liver disease, it is not surprising that bariatric surgery has been considered as a potential treatment for nonalcoholic fatty liver disease.
Only one study has reported the effects of bariatric surgery on nonalcoholic fatty liver disease and has demonstrated that fat and inflammation decrease, and progression from mild fibrosis to more severe fibrosis is arrested. (Patients with more severe fibrosis were not studied and, therefore it is not known if the progression from more severe fibrosis also is arrested.)
Nevertheless, because appropriate studies to determine both the benefits and risks of bariatric surgery have not been done, it is recommended that bariatric surgery should not be used as a treatment for NASH. Instead, patients should be selected for surgery irrespective of the presence or absence of NASH.
Liver transplantation
Once a liver has become cirrhotic and complications have developed, the options for treatment are either treating the complication as they arise or to replace the diseased liver with a transplanted liver. In fact, NASH has become the third most common cause of transplanting livers, only surpassed by alcoholic liver disease and hepatitis C, but it is expected to climb to the number one position due to the epidemic of obesity and diabetes. Unfortunately, NASH recurs frequently in the transplanted liver and then progresses to cirrhosis, presumably because the underlying causes – obesity and diabetes continue.
Homeopathic Treatment of Non-Alcoholic Fatty Livers
Chelidonium Majus
A prominent liver remedy, jaundice, pain under right scapula, Jaundice due to hepatic and gallbladder obstruction. Gall-colic. Distention. Fermentation and sluggish bowels. Constriction across, as by a string. Liver enlarged. Gallstones. Tongue yellow, with imprint of teeth; large and flabby. Taste bitter, pasty. Bad odor from mouth.
Constipation; stools hard, round balls, like sheep’s dung, bright yellow, pasty; clay-colored, stools float in water; alternation of diarrheas and constipation. Burning and itching of anus.
Urine: Profuse, foaming, yellow urine, like beer dark, turbid.
Pain in arms, shoulders, hands, tips of fingers. Icy coldness of tips of fingers; wrists sore, tearing in metacarpal bones. Whole flesh sore to touch. Rheumatic pain in hips and thighs; intolerable pains in heels.
Skin: Dry heat of skin; itches, yellow. Painful red pimples and pustules. Old, spreading, offensive ulcers. Wilted skin. Sallow, cold, clammy.
Cardus Marianus
The action of Cardus Marianus is centered in the liver, and portal system, causing soreness, pain, jaundice. Has specific relation to the vascular system. Abuse of alcoholic beverages, especially beer. Varicose veins and ulcers. Diseases of miners, associated with asthma. Dropsical conditions depending on liver disease, and when due to pelvic congestion and hepatic disease. Disturbs sugar metabolism. Influenza when liver is affected. Debility. Hemorrhages, especially connected with hepatic disease, dropsical accumulation of water in abdomen.
The next field is bleeding consequent to liver damage, liver pain and sensitiveness, feel fatigued or tired and have bouts of nausea and vomiting due to Liver Cirrhosis. Taste bitter. Aversion to salt meat. Appetite small; tongue furred; nausea; retching; vomiting of green, acid fluid. Stitches in left side of stomach, near spleen. Gallstone disease with enlarged liver.
Pain in region of liver. Left lobe very sensitive. Fullness and soreness, with moist skin. Constipation; stools hard, difficult, knotty; alternates with diarrhea. Stools bright yellow. Swelling of gallbladder with painful tenderness. Hyperemia of liver, with jaundice. Cirrhosis, with dropsy.
Rectum: Hemorrhagic piles, prolapse or rectum, burning pain in anus and rectum, hard and knotting, clayey stools. Profuse diarrhea due to rectal cancer. 10 drops doses .
Urine: Cloudy; golden-colored.
Skin: Itching on lying down at night. Varicose ulcers. Eruption on lower part of sternum.
Extremities: Pain in hip-joint, spreading through buttocks and down thigh; worse from stooping. Difficult rising. Weakness felt in feet, especially after sitting.
Phosphorus
Feels cold. Sharp, cutting pains. A very weak, empty, gone sensation felt in whole abdominal cavity. 
Liver congested. Acute hepatitis. Fatty degeneration. Jaundice. Pancreatic disease. Large, yellow spots on abdomen. Hunger soon after eating. Sour taste and sour eructation after every meal. Belching large quantities of wind, after eating. Throws up ingests by the mouthfuls. Vomiting: water is thrown up as soon as it gets warm in the stomach. Tibia inflamed and became necroes. Arms and hands become numb. Can lie only on right side. Post-diphtheritic paralysis, with formication of hands and feet. Joints suddenly give way.
Stool: Very fetid stools and flatus. Long, narrow, hard, like a dog’s. Difficult to expel. Desire for stool on lying on, left side. Painless, copious debilitating diarrhea. Green mucus with grains like sago. Involuntary; seems as if anus remained open. Great weakness after stool. Discharge of blood from rectum, during stool. White, hard stools. Bleeding hemorrhoids.
Urine: Hematuria, especially in acute Bright’s disease. Turbid, brown, with red sediment.
Fever: Chilly every evening. Cold knees at night. A dynamic with lack of thirst, but unnatural hunger. Hectic, with small, quick pulse; viscid night-sweats. Stupid delirium. Profuse perspiration.
Skin: Wounds bleed very much, even if small; they heal and break out again. Jaundice. Little ulcer outside of large ones. Petechiae. Ecchymosis. Purpura hemorrhagic. Scurvy. Fungus haematodes and excrescences.
Nux vomica
Bruised soreness of abdominal walls. Flatulent distension, with spasmodic colic. Colic from uncovering. Liver engorged, with stitches and soreness. Colic, with upward pressure, causing short breath, and desire for stool. Sour taste, and nausea in the morning, after eating. Weight and pain in stomach; worse, eating, Sour, bitter eructation. Nausea and vomiting.
Constipation, with frequent ineffectual urging, incomplete and unsatisfactory; feeling as if part remained un expelled. Alternate constipation and diarrhea-after abuse of purgatives. Urging to stool felt throughout abdomen. Itching, blind hemorrhoids, with ineffectual urging to stool; very painful; after drastic drugs. Diarrhea after a debauch; worse, morning. Frequent small evacuations. Scanty stool, with much urging. Dysentery: stools relieve pains for a time. Constant uneasiness in rectum. Diarrhea, with jaundice.
Skin: Body burning hot, especially face; yet cannot move or uncover without feeling chilly. Urticaria, with gastric derangement. Acne; skin red and blotchy.
Fever: Cold stage predominates. Paroxysms anticipate in morning. Excessive rigor, with blueness of fingernails. Aching in limbs and back, and gastric symptoms. Chilly; must be covered in every stage of fever. Perspiration sour; only one side of body. Chilliness on being uncovered, yet he does not allow being covered. Dry heat of the body.
Calcarea Carbonicum
This great Hahnemannian anti-psoric is a constitutional remedy par excellence. Its chief action is centered in the vegetative sphere, impaired nutrition being the keynote of its action, the glands, skin, and bones, being instrumental in the changes wrought. Increased local and general perspiration, swelling of glands, scrofulous and rachitic conditions,
craving for indigestible things-chalk, coal, pencils; also, for eggs, salt and sweets. Milk disagrees. Frequent sour eructation; sour vomiting. Dislike of fat. Loss of appetite when overworked. Heartburn and loud belching. Cramps in stomach; worse, pressure, cold water. Ravenous hunger. Swelling over pit of stomach, like a saucer turned bottom up. Repugnance to hot food. Pain in epigastric region to touch. Thirst; longing for cold drinks. Aggravation while eating. Hyperchlorhydria. Pain between shoulder-blades, impeding breathing. Cold knees cramps in calves. Sour foot-sweat. Weakness of extremities. Swelling of joints, especially knee. Burning of soles of feet. Fever with sweat. Pulse full and frequent. Chilliness and heat. Partial sweats. Night sweats, especially on head, neck and chest. Hectic fever. 
Abdomen sensitive to slightest pressure. Liver region painful when stooping. Cutting in abdomen; swollen abdomen. Incarcerated flatulence. Inguinal and mesenteric glands swollen and painful. Cannot bear tight clothing around the waist. Distention with hardness. Gall-stone colic. Increase of fat in abdomen. Umbilical hernia. Trembling; weakness, as if sprained. Children are late in learning to walk.
Stool: Crawling and constriction in rectum. Stool large and hard; whitish, watery, sour. Prolapse ani, and burning, stinging hemorrhoids. Diarrhea of undigested, food, fetid, with ravenous appetite. Children’s diarrhea. Constipation; stool at first hard, then pasty, then liquid.
Urine: Dark, brown, sour, fetid, abundant, with white sediment, bloody. Irritable bladder. Enuresis.
Skin: Unhealthy; readily ulcerating; flaccid. Small wounds do not heal readily. Glands swollen. Nettle rash; better in cold air. Warts on face and hands. Petechial eruptions. Chilblains. Boils.
Lycopodium
Corresponds to Grau ogle’s carbo-nitrogenous constitution, the non-eliminative lithaemic. Lycopodium is adapted more especially to ailments gradually developing, functional power weakening, with failures of the digestive powers, where the function of the liver is seriously disturbed. Atony. Malnutrition. Mild temperaments of lymphatic constitution, with catarrhal tendencies; older persons, where the skin shows yellowish spots, earthy complexion, uric acid diathesis, etc; also precocious, weakly children.
Dyspepsia due to farinaceous and fermentable food, cabbage, beans, etc. Excessive hunger. Aversion to bread, etc. Desire for sweet things. Food tastes sour. Sour eructation. Great weakness of digestion. Bulimia, with much bloating. After eating, pressure in stomach, with bitter taste in mouth. Eating ever so little creates fullness. Wakes at night feeling hungry. Hiccough. Incomplete burning eructation rise only to pharynx there burn for hours. Likes to take food and drink hot. Sinking sensation; worse night. kidney affections, red sand in urine, pain in renal region; worse before urination. Intolerant of cold drinks. Best adapted to persons intellectually keen, but of weak, muscular power.
Deep-seated, progressive, chronic diseases. Carcinoma. Emaciation. Debility in morning. Marked regulating influence upon the glandular (sebaceous) secretions. Pre-senility. Ascites lacks vital heat (immune system); has poor circulation, cold extremities. Pains come and go suddenly. Sensitive to noise and odors. Cirrhosis of Liver when the liver has atrophied due to long-standing Cirrhosis. Hepatitis may be predominantly present.
Abdomen is bloated, full. Constant sense of fermentation in abdomen, like yeast working; upper left side. Hernia, right side. Liver sensitive. Brown spots on abdomen. Dropsy, due to hepatic disease. Hepatitis, atrophic from of nutmeg liver.
Stool: Diarrhea. Inactive intestinal canal. Ineffectual urging. Stool hard, difficult, small, incomplete. Hemorrhoid; very painful to touch, aching.
Urine: Pain in back before urinating; ceases after flow; slow in coming, must strain. Retention. Polyuria during the night. Heavy red sediment.
Arsenic Album 
A profoundly acting remedy on every organ and tissue. Its clear-cut characteristic symptoms and correspondence to many severe types of disease make its homeopathic employment constant and certain.
Liver Cirrhosis with fatigue as the main symptom. The patient feels totally exhausted from doing a little labor. Cannot bear the sight or smell of food. Great thirst; drinks much, but little at a time. Nausea, retching, vomiting, after eating or drinking. Anxiety in pit of stomach. Burning pain. Craves acids and coffee. Heartburn: gulping up of acid and bitter substances which seem to excoriate the throat. Long-lasting eructation. Vomiting of blood, bile, green mucus, or brown-black mixed with blood. Stomach extremely irritable; seems raw, as if torn. Gastralgia from slightest food or drink. Dyspepsia from vinegar, acids, ice-cream, ice-water, tobacco. Terrible fear and dyspnea, with gastralgia; also, faintness, icy coldness, great exhaustion. Malignant symptoms. Everything swallowed seems to lodge in the esophagus, which seems as if closed and nothing would pass. Ill effects of vegetable diet, melons, and watery fruits generally. Craves milk.
Gnawing, burning abdominal pains; relieved by heat. Liver and spleen enlarged and painful. Ascites and anasarca. Abdomen swollen and painful. Pain as from a wound in abdomen on coughing.
Rectum: Painful, spasmodic protrusion of rectum. Tenesmus. Burning pain and pressure in rectum and anus.
Stool: Small, offensive, dark, with much prostration. Worse at night, and after eating and drinking; from chilling stomach, alcoholic abuse, spoiled meat. Dysentery dark, bloody, very offensive. Cholera, with intense agony, prostration, and burning thirst. Body cold as ice. Hemorrhoids burn like fire, relieved by heat. Skin excoriated about anus.
Urine: Scanty, burning, involuntary. Bladder as if paralyzed. Albuminoids. Epithelial cells; cylindrical clots of fibrin and globules of pus and blood. After urinating, feeling of weakness in abdomen. Bright’s disease. Diabetes.
Nux Vomica
Nux Vomica is greatest of polychrest. Liver Cirrhosis who has a history of long-term alcoholic abuse, chronic acidity and constipation. Sour mouth taste, and nausea in the morning, after eating. Weight and pain in stomach; worse, eating, sometime after. Flatulence and pyrosis. Sour, bitter eructation. Nausea and vomiting, with much retching. Ravenous hunger, especially about a day before an attack of dyspepsia. Region of stomach very sensitive to pressure. Epigastrium bloated, with pressure s of a stone, loves fats and tolerates them well. Dyspepsia from strong tea and/or coffee. Difficult belching of gas. Wants to vomit but cannot. soreness or stitching pain in liver region.
Inclination to take highly spicy diet, stimulants (tea, coffee, alcoholic drinks and fat etc). Mentally anger some and irritable nature, being sensitive to external impressions like noise and odor and also, a sensitivity to cold air.
Bruised soreness of abdominal walls. Flatulent distension, with spasmodic colic. Colic from uncovering. Liver engorged, with stitches and soreness. Colic, with upward pressure, causing short breath, and desire for stool. Weakness of abdominal ring region. Strangulated hernia. Forcing in lower abdomen towards genitals. Umbilical hernia of infants.
Stool: Constipation, with frequent ineffectual urging, incomplete and unsatisfactory; feeling as if part remained un expelled. Constriction of rectum. Irregular, peristaltic action; hence frequent ineffectual desire, or passing but small quantities at each attempt. Absence of all desire for defecation is a contra-indication. Alternate constipation and diarrhea-after abuse of purgatives. Urging to stool felt throughout abdomen. Itching, blind hemorrhoids, with ineffectual urging to stool; very painful; after drastic drugs. Diarrhea after a debauch; worse, morning. Frequent small evacuations. Scanty stool, with much urging. Dysentery: stools relieve pains for a time. Constant uneasiness in rectum. Diarrhea, with jaundice.
Urine: Irritable bladder; from spasmodic sphincter. Frequent calls; little and often. Hematuria. Ineffectual urging, spasmodic and strangury. Renal colic extending to genitals, with dribbling urine. While urinating, itching in urethra and pain in neck of bladder.
Cholesternium
Cancer of the liver. Obstinate hepatic engorgements. Burning pain inside; on walking holds his hand on side, hurts him so. Opacities of the vitreous. Jaundice; gallstones.
Cholesternium is the physiological opponent of Lecithin. Both seem to play role in the growth of tumors. Gallstones and insomnia.
Apocynum Cannabinum
Increases secretions of mucous and serous membranes and acts on cellular tissue, reducing edema and dropsy.
Nausea, with drowsiness. Thirst on walking. Excessive vomiting. Food or water is immediately ejected. Dull, heavy, sick feeling. Oppression in epigastrium and chest, impeding breathing. Sensation of sinking in stomach. Abdomen bloated. Ascites.
Stool: Watery, flatulent, with soreness in anus; worse after eating. Feeling as if sphincter were open and stools ran right out.
Urine: Bladder much distended. Turbid, hot urine, with thick mucus and burning in urethra, after urinating. Little expulsive power. Dribbling. Strangury. Renal Dropsy.
Quassia Amara
Acts on gastric organs as a tonic. Seems to possess marked action on eyes, producing amblyopia and cataract. Pain in right intercostal muscles above the liver. Pressure and stitches in liver, and sympathetically in spleen.
Stomach: Atonic dyspepsia, with gas and acidity. Heartburn and gastralgia. Regurgitation of food. Abdomen feels empty and retracted. Dyspepsia after infectious diseases; especially grip, dysentery. Tongue dry or with brown sticky coating. Cirrhosis of liver with ascites.
Urinary: Excessive desire-impossible to retain urine; copious micturition day and night. As soon as the child wakes up the bed is drenched.
Inclination to yawn and stretch. Sensation of coldness over back. Prostration, with hunger. Cold extremities, with sensation of internal coldness.
Bryonia Alba
Liver region swollen, sore, tensive. Burning pain, stitches; worse, pressure, coughing, breathing. Tenderness of abdominal walls. Pulse full, hard, tense, and quick. Chill with external coldness, dry cough, stitches. Internal heat. Sour sweat after slight exertion. Easy, profuse perspiration. Rheumatic and typhoid marked by gastro-hepatic complications. Hot swelling of feet. Joints red, swollen, hot, with stitches and tearing; worse on least movement. Every spot is painful on pressure.
Nausea and faintness when rising up. Abnormal hunger, loss of taste. Thirst for large draughts. Vomiting of bile and water immediately after eating. Worse, warm drinks, which are vomited. Stomach sensitive to touch. Pressure in stomach after eating, as of a stone. Soreness in stomach when coughing. Dyspeptic ailments during summer heat. Sensitiveness of epigastrium to touch.
Skin: Yellow; pale, swollen, dropsical; hot and painful. Seborrhea. Hair very greasy.
China Officianalis
Much flatulent colic; better bending double. Tympanite abdomen. Pain in right hypochondrium. Gall-stone colic. Liver and spleen swollen and enlarged. Jaundice. Internal coldness of stomach and abdomen. Vomiting of undigested food. Slow digestion. Weight after eating. Darting pain crosswise in hypogastric region. Flatulence: belching of bitter fluid or regurgitation of food gives no relief.
Stool: Undigested, frothy, yellow; painless; worse at night, after meals, during hot weather.
Berberis Vulgaris
Stitches in region of gallbladder; worse, pressure, extending to stomach. Catarrh of the gallbladder with constipation and yellow complexion. Stitching pain in front of kidneys extending to liver, spleen, stomach, groins, Poupart’s ligament. Sticking deep in ilium.
Stool: Constant urging to stool. Diarrhea painless, clay-colored, burning, and smarting in anus and perineum.
Eupatorium Perfoliate
Tongue yellow. Taste bitter. Hepatic region sore. Great thirst. Vomiting and purging of bile, of green liquid several quarts at a time. Vomiting preceded by thirst. Hiccough. Avoids tight clothing.
Stool: Frequent, green watery. Cramps. Constipated, with sore liver.
Iodium 
Throbbing at pit of stomach. Ravenous hunger and much thirst. Empty eructation, as if every particle of food were turned into gas. Anxious and worried if he does not eat. Loss flesh, yet hungry and eating well. Flushes of heat all over body. Marked fever, restlessness, red cheeks, apathetic. Profuse sweat.
Heart feels squeezed. Myocarditis, painful compression around heart. Palpitation from least exertion. Tachycardia.
Liver and spleen sore and enlarged. Jaundice. Mesenteric glands enlarged. Pancreatic disease. Cutting pain in abdomen.
Skin: Hot, dry, yellow and withered. Glands enlarged. Nodosities. Anasarca of cardiac disease.
Stool: Hemorrhage at every stool. Diarrhea, whitish, frothy, fatty. Constipation, with ineffectual urging; better by drinking cold milk. Constipation alternating with diarrhea.
Urine frequent and copious, dark yellow green (Bovista), thick, acrid with cuticle on surface.
Ammonium Muriaticum
Thirst for lemonade, regurgitation of food, bitter water brash. Nausea. Gnawing in stomach. Epigastric pain immediately after eating. Cancer of stomach.
Abdomen: Splenic stitches, especially in the morning, with difficult breathing. Pain around navel. Abdominal symptoms appear during pregnancy. Chronic congestion of liver. Excessive fatty deposit around abdomen. Much flatus. Strained feeling in groin.
P. S: This article is only for doctors having good knowledge about Homeopathy and allopathy, for learning purpose(s).
For proper consultation and treatment, please visit our clinic.
None of above-mentioned medicine(s) is/are the full/complete treatment, but just hints for treatment; every patient has his/her own constitutional medicine.
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Dr. Sayyad Qaisar Ahmed (MD {Ukraine}, DHMS), Abdominal Surgeries, Oncological surgeries, Gastroenterologist, Specialist Homeopathic Medicines.
Senior research officer at Dnepropetrovsk state medical academy Ukraine.
Location: Al-Haytham clinic, Umer Farooq Chowk Risalpur Sadder (0923631023, 03119884588), K.P.K, Pakistan.
Find more about Dr Sayed Qaisar Ahmed at:
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